The heterozygous mutations
of SLC26A8 are not the main actors but might be the guest players
for male infertility
Mohan Liu1,2, Jinhui Li3, Yaqian
Li1, Chuan Jiang1, Wenming
Xu1 *, Yihong Yang4*, Ying
Shen1 *
1 Department of Obstetrics/Gynecology, Key
Laboratory of Obstetric, Gynecologic and Pediatric Diseases and Birth
Defects of Ministry of Education, West China Second University Hospital,
Sichuan University, Chengdu 610041, China
2 State Key Laboratory of Biotherapy and Cancer
Center, West China Hospital, Sichuan University and Collaborative
Innovation Center, Chengdu 610041, China.
3Department of Neonatology, Key Laboratory of
Obstetric, Gynecologic and Pediatric Diseases and Birth
Defects of Ministry of Education, West China Second University Hospital,
Sichuan University, Chengdu 610041, China
4 Reproduction Medical Center of West China Second
University Hospital, Key Laboratory of Obstetric, Gynecologic and
Pediatric Diseases and Birth Defects of Ministry of Education, Sichuan
University, Chengdu 610041, China
Mohan Liu, Jinhui Li and Yaqian Li contributed equally to this work.
*Correspondence should be addressed to: Wenming Xu, Department of
Obstetrics/Gynecology, Key Laboratory of Obstetric, Gynecologic and
Pediatric Diseases and Birth Defects of Ministry of Education, West
China Second University Hospital, Sichuan University, Chengdu 610041,
China, E-mail: xuwenming@scu.edu.cn; Yihong Yang, Reproduction Medical
Center of West China Second University Hospital, Key Laboratory of
Obstetric, Gynecologic and Pediatric Diseases and Birth Defects of
Ministry of Education, Sichuan University, Chengdu 610041, China E-mail:
yyhpumc@foxmail.com; Ying Shen, Department of Obstetrics/Gynecology, Key
Laboratory of Obstetric, Gynecologic and Pediatric Diseases and Birth
Defects of Ministry of Education, West China Second University Hospital,
Sichuan University, Chengdu 610041, China, E-mail:
yingcaishen01@163.com.