Introduction
Severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) infection
is a major public health problem, with hospitalizations, admissions to
the intensive care unit (ICU), and even deaths Compared with
non-pregnant women of childbearing age with SARS-CoV-2 infection,
pregnant women are more likely to be needed for respiratory support and
admitted to an ICU [2]. Pregnant women with SARS-CoV-2 infection are
at high risk for adverse perinatal outcomes such as early pregnancy
loss, fetal growth retardation, and preterm delivery [3,4].
Impaired placental function, hypoperfusion, and inflammation might lead
to fetal decompensation that increased risk of perinatal mortality and
morbidity [5,6].
Cardiac output depends on heart rate, preload, afterload and myocardial
contractility . An increase in afterload or a decrease in preload causes
a decrease in cardiac output Changes in cardiac output have been
demonstrated in hydrops fetalis, fetal growth retardation (FGR), anemia,
and various pathological conditions . In this present study, we aimed to
evaluate changes of fetal cardiac output in pregnant women who recovered
from SARS-Cov-2 infection.