Introduction
Severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) infection is a major public health problem, with hospitalizations, admissions to the intensive care unit (ICU), and even deaths Compared with non-pregnant women of childbearing age with SARS-CoV-2 infection, pregnant women are more likely to be needed for respiratory support and admitted to an ICU [2]. Pregnant women with SARS-CoV-2 infection are at high risk for adverse perinatal outcomes such as early pregnancy loss, fetal growth retardation, and preterm delivery [3,4].
Impaired placental function, hypoperfusion, and inflammation might lead to fetal decompensation that increased risk of perinatal mortality and morbidity [5,6].
Cardiac output depends on heart rate, preload, afterload and myocardial contractility . An increase in afterload or a decrease in preload causes a decrease in cardiac output Changes in cardiac output have been demonstrated in hydrops fetalis, fetal growth retardation (FGR), anemia, and various pathological conditions . In this present study, we aimed to evaluate changes of fetal cardiac output in pregnant women who recovered from SARS-Cov-2 infection.