Conclusion
This study revealed differences in the metabolic profiles of vaginal
secretions in patients with VVC due to C. albicans infection and in
healthy people. These differentially expressed metabolites may be
involved in the VVC process. The analysis results showed that
significantly elevated LA levels in VVC could bind to ACSL1, suggesting
that LA may exert its biological function through the MAPK and NF-κB
signaling pathways. Our results provide new insights into the diagnosis
and treatment of VVC, and further research is needed to verify the
mechanism of LA in VVC.