Conclusion
This study revealed differences in the metabolic profiles of vaginal secretions in patients with VVC due to C. albicans infection and in healthy people. These differentially expressed metabolites may be involved in the VVC process. The analysis results showed that significantly elevated LA levels in VVC could bind to ACSL1, suggesting that LA may exert its biological function through the MAPK and NF-κB signaling pathways. Our results provide new insights into the diagnosis and treatment of VVC, and further research is needed to verify the mechanism of LA in VVC.