CONCLUSION
In summary, our study
demonstrated that Empagliflozin effectively ameliorated HFD/STZ-induced
liver fibrosis in T2DM mice through increasing cecal relative abundance
of Lactobacillus and reducing the abundance ofRuminococcus and Adlercreutzia , which might be a novel
therapeutic direction for liver fibrosis (Supplementary Figure 4).
MATERIALS AND METHODS